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[DOI] [PMC free article] [PubMed] [Google Scholar] PRISMA [WWW Document], n.d

It is conceivable that this general mechanism can explain Mercury-induced oxidative stress not only through a continuous depletion of the reduced glutathione pool, but also by interfering with enzymes operating through active sites based on vicinal thiol groups or which are regulated by glutathionylation of sensing cysteine residues

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