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Therefore, BKTD should be considered not only as a defect in ketone body utilization but also as a defect in isoleucine catabolism with the potential for insidious cerebral toxicity 1

Furthermore, the effect of methionine is tissue-specific, and studies that measured the correlation between methionine intake and plasma s-adenosyl methionine to s-adenosyl homocysteine ratio have yielded inconsistent findings

OECD (2015) Test no

Ann Nutr Metab 2013;62:11322

Cell proliferation induced by GPE was further delineated by an increased level of phosphorylated Akt, indicating potential upregulation of the ERK ( Figure 6 ) and PI3K/Akt signaling pathways ( Figure 7 ) pathways, which are among the signaling pathways involved in the regulation of cell proliferation