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Description
This review focuses on the expression, regulation and evolution of GPX3 in non-neoplastic diseases, assesses its potential as a prognostic and diagnostic biomarker and explores new therapeutic avenues targeting GPX3
Adding semaglutide or tirzepatide adds redundant GLP-1 (and GIP, in the case of tirzepatide) activity without new pathway coverage
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Curcumin has capacity of regulating expression and activity of P-gp and by suppressing its activity, curcumin prevents drug resistance in tumor cells
It upregulates the Anti-Inflammatory Gene Transcription Factor, and Human Growth Hormone receptors, all leading to better systemic repair response
