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Without any clear rationale, the study outcome measures were purposefully modified midtrial to alter the findings in favour of their hypothesis (549, 550)

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32% HCD collision energy was used for MS2 fragmentation

Consequently, neuroinflammation in multiple sclerosis originates from a chronic pro-inflammatory state caused by intrinsic metabolic defects in macrophages, while post-stroke inflammation emphasizes the dynamic transition of polarization phenotypes during the acute phase

Authoritative third party test reports are available