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Although the genetic variations in the genes of astrocytes or microglia have not been reported, the knockdown of chemokine receptor 1 ( CX3CR1 ) resulted in a reduction in microglia, ASD-associated functional connectivity, and behavioral deficits and defects in synaptic pruning [200, 201]

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The findings revealed that higher concentrations of PSNPs led to significant adverse effects, including alterations in blood parameters and metabolic enzymes, increased stress indicators like cortisol and glucose levels, and notable histopathological changes in gill and liver tissues

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