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Diabetes-associated excess kidney tissue RAS activation increases angiotensin II-mediated glomerular efferent arteriolar vasoconstriction and glomerular hyperfiltration, tubular sodium reabsorption, and signalling through intracellular pathways, resulting in inflammation and oxidative stress [13]
10.1155/2014/919256 102 TomlinsonB.BompartF.GrahamB
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Br J Anaesth 126(3):567571
Mohammadyari F, et al
