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Description
GLP-1 medications mimic the GLP-1 incretin hormone that the body produces
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34 In our study, normal CSMCs were capable of producing TNF-, while LPS enhanced this capability and GLP-1 co-treatment significantly inhibited its expression and secretion, suggesting an important role for CSMC-driven TNF- in the pathogenesis of gastrointestinal inflammatory diseases through recruitment and activation of different inflammatory cells
The GLP-1 receptor agonist dulaglutide attenuates hepatic steatosis in obesity via a weight-independent mechanism
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