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This additional sensitivity analysis was performed because of the well-known tumor-promoting potential of insulin (42,43)

To sustain its momentum, the GLP-1 revolution will require pharmadevice collaborations, analytical innovation, and sustainable design, which will be key to tackling the growing device bottleneck

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Abstract The glucagon-like peptide-1 receptor (GLP-1R) and the glucagon receptor (GCGR) are members of the secretin-like class B family of G-protein-coupled receptors (GPCRs) and have opposing physiological roles in insulin release and glucose homeostasis 1