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Additionally, evidence indicates that GLP-1 enhances endoplasmic reticulum (ER)-mitochondria interactions, leading to increased mitochondrial activity [38]

doi:10.3390/nu15163583

Clinicians must recognize that GLP-1 receptor agonists are not FDA-approved for treating alcohol use disorder or reducing alcohol cravings

The GLP-1 receptor agonist component is considered, based on established class effects, a likely contributor to these gastrointestinal adverse events, as GLP-1 activation slows gastric emptying and alters intestinal transit time

This unique metabolic pathway aligns with the specificity of CGRP inhibitors, minimizing interactions with hepatic enzymes and potential drug-drug interactions