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Immunofluorescence results showed that EA effectively activated GLP-1 neurons in the NTS, and the number of activated GLP-1 neurons decreased significantly after chemogenetic suppression, demonstrating that the chemogenetic tools virus was functioning properly (Fig

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(44) reported that both GIPR agonist (GIPRA) and dual GIP/GLP-1 receptor agonist similarly improved glucose tolerance in GLP-1R knockout mice, but not in dual incretin receptor knockout mice