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However, the anatomic distribution of the L-cells (highest number in distal gut) and the rapid increase in postprandial circulating GLP-1 (35) that occurs before nutrients reach the distal gut (8, 63) is evidence supportive of neural, endocrine and/or paracrine mechanisms being more critical to nutrient-induced increases in circulating GLP-1 (164)

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2 GLP-1 release also slows gastric emptying, reduces appetite, and decreases food intake, thus making this peptide an attractive target for obesity pharmacologics