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Chronic inflammation in type 2 diabetes (T2D), characterized by constitutively activated immune cells and elevated pro-inflammatory mediators along with hyperglycaemia and increased free fatty acids and branched chain amino acid levels, significantly alters the immuno-metabolic axis

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Because GCGR activation is associated with changes in hepatic energy metabolism and lipid oxidation pathways, GLP-RTprovides a framework for studying how multiple endocrine signaling inputs influence whole-body energy partitioning in controlled experimental environments [5]

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