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1,2 For this reason, its important to further understand the mechanism of GLP-1 therapies, said Lorenzo Leggio, a neuroendocrinologist at the National Institute on Drug Abuse who mediated the GLP-1 press conference at the 2025 Society for Neuroscience meeting

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Insulin sensitivity changes

The nervous system has fundamentally misinterpreted a harmless signal as dangerous, and the brain responds with genuine pain

This effect, combined with the negative energy balance driven by low food intake of a dual GLP-1R/GIPR agonist, promotes a shift to lipid catabolism, as noted repeatedly in rodent studies