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This is actually one of its therapeutic mechanisms, slowing how quickly food leaves the stomach helps reduce post-meal blood sugar spikes and promotes feelings of fullness

Gastrointestinal effects represent the most common concern

It is able to work in a similar way to our naturally occurring GLP-1 in the body and therefore has very similar effects including: Increase release of insulin when blood sugar is elevated

If semaglutide continues to show promise in this area, it could lead to a new approach for addressing multiple substance use behaviors at once

Based on the statements above, it seems like HIMS compounded GLP-1 business is in a particularly vulnerable situation