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For this, we target intracellular and membrane-bound proteins implicated in inflammatory pathways of AD
And this first result was followed a couple of months later by a similar report with a very similar set of results: Title: Glucagon-like peptide 1 receptor stimulation reverses key deficits in distinct rodent models of Parkinsons disease

They are often used in combination with other hypoglycemic agents like glycosidase inhibitors or insulin for better blood glucose management

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While Rascols leading hypothesis is that GLP-1 drugs are working to reduce inflammation, he acknowledges that the reductions in Parkinsons symptoms in his study could also be explained by something else
