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These results suggest that exendin-4-induced suppression may be mediated by a distinct G s /cAMP-PKA/IP 3 /Ca 2+ /CaM/CaMKII signaling pathway, following the activation of GLP-1Rs

Patients with a history of small-bowel obstruction or fistulizing disease should consult their gastroenterologist before initiating any GLP-1, as slowed gastric emptying and reduced motility may pose mechanical risks

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These agents indirectly influence the CA1 region of the ventral hippocampus, thereby modulating hunger and satiety, as well as emotional responses to food, which are implicated in BEDs (BED) [23]