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in the other outcome, the injured tissue continues to deteriorate, and the mechanism of the persisting damage involves gliosis with the loss of support of neurotrophic factors, persistent inflammatory receptor activation, and changes in microglia and astrocytes that continue to release harmful cytokines, which promote neuronal death and axonal injury

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More than 100 preclinical research studies suggests that GLP-1 drugs could protect against cognitive decline

Estimated energy requirements are personalized based on age, male/female, and activity level, however when analyzing group data this is not feasible