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This early window reflects the drug's activation of GLP-1 receptors in your hypothalamus, the brain region controlling hunger signals and fullness cues

It helps the pancreas release insulin (only when glucose is present), suppresses glucagon, slows gastric emptying, and signals satiety to the brain

Nutrition-wise, create a 300-500-calorie deficit relative to your TDEE

Both have gained popularity due to their effectiveness, but its important to understand their differences and similarities to better prepare you for conversations with a licensed medical provider about whether these medications are right for you
