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According to the literature addressing clinical trials, GI AEs usually develop in 4070% of treated patients, although they have sometimes been reported in up to 85% [9,10,11,12,13,14,15,16,17]

On the other hand, limitations in available GLP-1RA choices have led to selection biases, particularly in the early 2010s and in regions where access to other GLP-1RAs was relatively delayed

Currently, the FDA is advising against the use of GLP1 agonists in people at risk for MTC or MEN2 syndrome ( N Engl J Med

The pathological hallmarks of the disease are the progressive and selective degeneration of the dopaminergic neurons in the substantia nigra, resulting in dopamine depletion in the striatum, and the presence of Lewy bodies in the remaining neurons [1]