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Mechanisms of Toxicity Image 2 (Source) The mechanism of toxicity for each aforementioned venom component will be described individually below

Unlike traditional single-pathway or dual-pathway incretin compounds, this molecule is structurally designed to activate GLP-1 (glucagon-like peptide-1), GIP (glucose-dependent insulinotropic polypeptide), and glucagon receptors simultaneously

By contrast, peptides such as PYY and apelin were discontinued primarily due to formulation limitations or tolerability at supraphysiologic dosespharmacologic rather than biological failuresand therefore meet criteria for re-evaluation

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