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The reason retatrutide adds GCGR activity is that glucagon receptor activation in the liver also drives fatty acid oxidation and, when combined with GLP-1 and GIP agonism, produces a metabolic phenotype that single- and dual-receptor agonism do not

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Mounting evidence suggests these GLP-1 receptor agonist medications may be linked to vision loss related to NAION and gastroparesis (stomach paralysis) and other severe injuries

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