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However, she continued, a recent paper showed that ablating the GLP1R-expressing neurons in the hindbrain dorsal vagal complex, including the nucleus of the solitary tract, blocks the ability of the systemic receptor agonist to suppress intake (Huang et al., 2024)

Clinical studies suggest that up to 39% of the weight lost on GLP-1 medications can come from lean body mass

Second, human research is still in the development stage

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The effects of the active forms of GLP-1 are mediated by a G protein-coupled receptor, GLP-1R, which is expressed in several sites, including enteric and vagal nerves, the stomach, pancreas, intestine, and various brain regions [15]