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Consistent with a role of vagal afferents and vagal afferent GLP-1 receptors in the eating-inhibitory effect of peripheral endogenous GLP-1, virus-mediated knockdown of GLP-1R in vagal afferent neurons increased meal size, accelerated gastric emptying, and reduced prandial insulin secretion in rats, while not affecting 24 h food intake or body weight [518]

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The molecule was rationally designed by Eli Lilly scientists to simultaneously engage three G-protein-coupled receptors central to metabolic homeostasis: the glucose-dependent insulinotropic polypeptide (GIP) receptor, the glucagon-like peptide-1 (GLP-1) receptor, and the glucagon receptor

[DOI] [PMC free article] [PubMed] [Google Scholar] 40.Teixeira L.B., Blouin M.J., Le Gouill C., Picard L.P., Costa-Neto C.M., Bouvier M., Parreiras-E-Silva L.T

Davies, M., D'Alessio, D