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This observation also highlights the complexity that modifying a residue within a cholesterol binding site might have on the overall pattern of receptor interactions with cholesterol and other plasma membrane lipids, as increased segregation to cholesterol-rich lipid rafts occurs despite an overall reduction in total cholesterol binding under vehicle conditions observed for the V229A mutant receptor

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Likely, the ability of GLP1RA to mitigate obesity and hyperglycemia partly accounts for their effects on the CNS

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Using standard endpoints to assess the metabolic impact of PERT (pancreatic enzyme replacement therapy) and its optimisation