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Under high-glucose and high-lipid stress, GLP-1RAs reduce apoptosis and mitigate mitochondrial dysfunction in human microvascular endothelial cells by inhibiting ERK1/2, JNK and p38 pathways, while suppressing caspase-3 and PARP cleavage (167)

Bile acids trigger GLP-1 release predominantly by accessing basolaterally located G proteincoupled bile acid receptors. Endocrinology 156.11 (2015): 3961-3970

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