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Further evidence seems to suggest a role for Tregs in improving immunopathologic damage of target organs in diabetic complications: for example, a study of diabetic nephropathy in db/db mice (Leptin-Receptor deficient mice) showed that Tregs could attenuate T2D-related kidney morphologic and functional lesions (5)

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Nevola R, Epifani R, Imbriani S, et al

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