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The present study aimed to elucidate whether DPP4 and TGF- serve crucial roles in regulating the EMT process and tissue fibrosis, which can result in PF and, ultimately, PD failure

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The ability of metformin to increase GLP-1 levels independently of effects on DPP-4 suggests there could be a direct benefit in terms of incretin action by combining metformin with DPP-4 inhibitors

The effect is most noticeable during the active dose-escalation phase and the first few weeks at target dose

lipoproteins and endotoxins) has been confirmed using HEK-Blue TLR2 and HEKBlue TLR4 cells