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No current therapy fully meets these criteria, particularly in improving fibrosis4

Note: These trials differ in design, duration, and populations, so comparisons are approximatenot head-to-head

This represents a direct pharmacological effect rather than an indirect consequence of diabetes control

GLP-1 and NMDA receptors are expressed throughout the mammalian brain and, although reports indicate that GLP-1 analogues engage central appetite circuits through distinct subpopulations of neurons (most notably in the brainstem and the hypothalamus) 36 , feedforward neural pathways modulated by inhibition of NMDA receptors specifically in GLP-1-receptor-positive neurons may differ from those engaged by exclusive activation of GLP-1 receptors

Preliminary research indicates that the activation of GLP-1R facilitates mechanisms essential for telomere stability, potentially delaying cellular aging and extending cellular lifespan