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The Journal of nutrition, 136(6), pp.1533-1538

doi: 10.1111/dom.12596

This, in turn, stimulates downstream signaling elements dependent on both protein kinase A (PKA) and exchange protein directly activated by cAMP (EPAC) activation, which triggers the opening of voltage-dependent calcium channels (VDCCs), resulting in insulin exocytosis [24, 25]

Although the totality of the data that have been reviewed provides reassurance, pancreatitis will continue to be considered a risk associated with these drugs until more data are available

An exception is glicentin and its cleavage product oxyntomodulin, the concentrations of which need to be combined to match the amount of secreted GLP-1 [41,53]