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A recent study demonstrated that GLP-1 enhances preingestive satiety through hypothalamic circuits in both mice and humans, with GLP-1R-expressing neurons in the dorsomedial hypothalamus (DMH) emerging as key candidates for encoding this satiety signal [24]

These findings should not be interpreted as an assertion that the DVC is the only CNS site mediating the intake suppressive effects of GLP-1- and amylin-based pharmacotherapy

[abstract] Mov Disord

Whether or not the GLP-1 boom reshapes alcohol entirely, its already reshaping how people think about what goes in their glass.

It is to work with a clinical team that takes side effects seriously and responds quickly