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Both studies examined the effect of GLP-1RA on fibroblast-like synoviocytes (FLSs), cells in the synovial fluid of joints that normally play a protective and homeostatic role in joint function but can transform into a pathological phenotype that is apoptosis-resistant and proinflammatory in function in the presence of inflammatory stimuli
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However, upregulation of GLP-1 signaling does not restore the number of glutamatergic neurons in BLA
