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Contact of nutrients with L-cells in the distal gut likely explains the prolonged duration of elevated GLP-1 during meals, and it might also be responsible for the typically observed increased levels of GLP-1 following gastric bypass and, albeit less so, after sleeve gastrectomy surgery [[211], [212], [213], [214], [215], [216]], because surgical repositioning of the distal gut more rapidly exposes the distal L-cells to incoming nutrients [144]
The rare bone condition known as Osteomalacia also occurred twice as often among GLP-1 users
