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Description
The current report aims to present an overview of current knowledge on GLP-1 RAs based on pragmatic review of the available clinical evidence
However, DPP-4 inhibitors have not reduced the risk of major adverse cardiovascular events, possibly because potentiation of SDF-1 enhances plaque growth and instability, activates deleterious neurohormonal mechanisms, and promotes cardiac inflammation and fibrosis
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In addition to GLP-1, linagliptin also increased postprandial plasma levels of GIP to a similar degree in all genotypes
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