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Indeed, we now understand that GLP-1 controls inflammation (75), reduces experimental kidney injury (76), and like GLP-2, acts as a potent intestinal growth factor through mechanisms including stimulation of crypt fission (ref

The Interaction of GLP-1 and Sulphonylureas The role of the SUR1 receptor in first-phase insulin secretion, and potentially in granule acidification required for second-phase secretion, provides a possible explanation for the observed clinical interaction between GLP-1R agonists and SUs

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