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Two (50%) participants were on one T2D medication and two (50%) were on two T2D medications

We reason that in chronic obesity, sustained GLP-1 activation in Tmprss2 / mice improves insulin sensitivity and glucose metabolism but, at the same time, paradoxically regulates insulin-induced GIP secretion independent of PAR2 signaling, thus superseding TMPRSS2 deficiency

Likelihood ratio tests were employed to compare differences between two groups, and Wald tests were utilized to compare differences between three or more groups

Addict Behav 1982;7(1):4755

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