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For an in-depth description of the biology of GLP-1, readers are referred to a recent review by Mller et al

2024 Jul 25;391(4):311-319.10.1056/NEJMoa2401755 [DOI] [PubMed] 42.Coskun T, Urva S, Roell WC, Qu H, Loghin C, Moyers JS, et al

subsequent preclinical studies have thus screened agonists with primates with Trp33 ECD or receptor mutants with substitution of Ser33 ECD by Trp33 ECD

In addition to anti-glucolipotoxic effects on the endothelium [120], treatment in Apoe / mice inhibits formation of the receptor for advanced glycosylation end-products (RAGE) in the aorta and lowers serum AGE levels [121]

Unmet Needs and Whitespace Opportunities Despite the remarkable clinical and commercial success of GLP-1 agonists, significant unmet needs persist that define the whitespace for next-generation innovation