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It is not widely appreciated, however, that in healthy individuals, gastric emptying of nutrients is regulated primarily by inhibitory neurohormonal feedback arising from their interaction with the small intestine, rather than intragastric mechanisms, and that both the length and region of small intestine exposed to nutrients modulate this feedback [12, 13]

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indeed, any augmentation of GLP-1 responses would probably be welcome and could help control glycaemia in patients with type 2 diabetes

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