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Interestingly, it has been shown that rats consuming a 3 g meal displayed significant elevations in plasma GLP-1 within 56 min only in the hepatoportal vein and not in the vena cava [78], suggesting that, under physiological conditions, GLP-1R within the CNS is only reached by brain-derived GLP-1, since GLP-1 is also expressed by a discrete population of caudal brainstem neurons localized to neurons within the caudal (visceral) nucleus of the solitary tract (cNST) and adjacent medullary reticular formation in rodents and primates, including humans [83,84,85,86]

8 Based on survey responses of 166 participants in a 6-month clinical trial who had overweight or obesity, and were taking semaglutide/tirzepatide for weight loss through Weight Watchers Clinic and provided the GLP-1 Success Program

The Human Side of the GLP-1 Divide Numbers only tell part of the story

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Figure S3: (A) Individual study result of primary outcome: incidence of hearing loss, (B) Individual study result of primary outcome: incidence of hearing loss in subgroup of dosage, (C) Individual study result of drop-out rate