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Description
These findings provide new insights into the pathogenesis of CM and suggest that microglial GLP-1R in the TNC may be a new target for the treatment of CM
Most of the small molecules now in development bind in this same general region and exploit the Trp33 anchor
Incretin effects of increasing glucose loads in man calculated from venous insulin and C-peptide responses
Plant-based compounds might have some similar effects, but their mechanism of action may be different
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