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Collectively, our data suggest that T3 might also be able to increase the production of non-12-OH FXR-antagonistic BA species in human likely consequent to the increased synthesis of non-12-OH BAs, thereby potentiating the GLP-1 and insulin production by suppressing the intestinal FXR signaling (Fig

3.1.5 Lactococcus lactis A primary species in probiotic research, specific strains like ML-2018 demonstrated potent anti-inflammatory activity by downregulating LPS-induced cytokines (65, 66)

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