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PPAR- is also strongly expressed in thyroid tissue of patients with thyroiditis and Graves disease and in the orbital tissue of patients with thyroid eye disease (TED) [116]

The most common secondary damage is ipsilateral thalamic degeneration after cerebral infarction, including axonal degeneration, neuronal loss, gliosis, -amyloid (A) deposits, and hyperphosphorylation of Tau protein (152, 153)