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[8,16] Once activated, Akt regulates several crucial downstream mechanisms, including glucose uptake through GLUT3/GLUT4 transporters, stabilization of neuronal membrane potential through Na + /K + -ATPase activity, modulation of GABAergic neurotransmission, and regulation of mitochondrial function through mammalian target of rapamycin (mTOR) and AMP-activated protein kinase (AMPK) pathways

250350 g) (Charles River Laboratories) were maintained in individual cages under controlled temperature (2123 C) and light (light on 08:0020:00) conditions

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However, clinically, a recent study suggests that patients on long-acting GLP1-RA have a significantly higher proportion of gastric residue in comparison to the non-GLP1-RA treatment group measured by EGD in patients with diabetes, indicating that the effect on gastric emptying even for a patient on a long-acting GLP1-RA may be significant [6]