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Haobin Yes team at Fudan University on their latest Cell Metabolism publication, revealing that dual targeting of SLC25A51 and SDH selectively depletes mitochondrial NAD+ to eradicate KRAS-driven AML, offering a new therapeutic paradigm based on metabolic vulnerability

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The first factor identified was gastric inhibitory polypeptide (GIP), which was thought to increase glucose-stimulated insulin secretion

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