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Increasing evidence suggests that alternative molecular mechanismssuch as tau pathology, neuroinflammation, mitochondrial dysfunction, brain insulin resistance, and vascular impairmentplay critical roles in disease progression

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See some of our original CardioNerds episodes to review the clinical manifestations, diagnosis and treatment of HCM

Effect of semaglutide treatment on a proteomic signature of secondary CVD risk Previous work generated and validated a 27-protein signature predicting a 4-year likelihood of myocardial infarction, stroke, heart failure or death in patients with established CVD 22

Level 2 or 3 hypoglycemic events should trigger a re-evaluation of the individuals diabetes treatment plan, with consideration of deintensification of therapy within individualized glycemic goals