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Semaglutide may possibly increase the expression of ubiquitin ligase parkin, leading to ubiquitination of mitochondrial proteins, effectively marking them for removal through mitophagy activation [137]

Diabetes Obes Metab (2017) 19(9):124251

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Moreover, due to these substitutions, semaglutide has a prolonged half-life of approximately 168 h, which is impressive given the knowledge that native GLP-1 has a half-life of 2 min, in comparison [98, 99]