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Dysgeusia also has been reported by people taking an oral form of semaglutide

Because gastric emptying is a prerequisite for glycemic rises after meals, which in turn trigger insulin secretion, deceleration of gastric emptying in response to exogenous GLP-1 leads to lesser post-meal glucose excursions and reduced insulin secretory responses [809]

Interaktionen GLP-1-Rezeptor-Agonisten verlangsamen die Magenentleerung und knnen so die Absorption und die Pharmakokinetik anderer Wirkstoffe beeinflussen
Targeted disruption of peptide transporter Pept1 gene in mice significantly reduces dipeptide absorption in intestine