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So what we do is we take a naturally occurring hormone, we take it out, synthesize it into pharmacologic amounts, give it back to individuals, so we're able to amplify these satiety signals that someone's not quite getting, so they feel full

demonstrated that PPG neurons in the brainstem mediate the reduction of food intake by lorcaserin and that the combination of lorcaserin with GLP-1RA resulted in additive effects on food intake potentially providing a strategy to increase the therapeutic margin of future 5-HT2CR agonists (121)

Only dulaglutide treatment did not decrease TDI, however this was a small study (n=18) (52)

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Preclinical studies have shown that large doses of GLP-1RAs can reduce lipid deposition and plaque volume on the aortic surface of hyperglycemic mice via an AMPK-independent action in streptozotocin (STZ)-induced hyperglycemic and hyperlipidemic mice or type 1 diabetic rats [55]