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The profound anatomical and pathophysiological disparities between these models and human disease create a significant translational gap. Acknowledging these limitations is key to interpreting preclinical data cautiously and designing more relevant experimental paradigms

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Los estudios clnicos han demostrado que la restauracin de los niveles de GLP-1 mediante agonistas del receptor de GLP-1 puede reducir la hemoglobina A1c en 0.8-1.5%, dependiendo del agente especfico y la dosis utilizada