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Defective p53 post-translational modification required for wild type p53 inactivation in malignant epithelial cells with mdm2 gene amplification

Given the difficulty of capturing the complete human smoking phenotype using a murine model, we used a simpler conditioned media exposure model to ascertain the effects of smoke exposure on the interaction between HNSCC cells and PBMCs

As a result, hepatocytes with de novo lipogenesis are prone to convert into HcPC, and the liver contains HcPC that undergoes multiple divisions to give rise to fully malignant HCC after long-term lipometabolic disturbance challenge 125

[DOI] [PubMed] [Google Scholar] 12.Pustisek N., Situm M

Implication of Ultraviolet Irradiation in Photo-carcinogenesis