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Epigenetic and transcriptomic analyses have identified the amplification of the MYC family proto-oncogenes ( MYC , MYCL and MYCN ) [136], with MYCL being associated with the ASCL1-subtype and MYC being associated with other subtypes [126]

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Both mono-and di-methylation of histone H3 residues (H3K27me2/3, H3K36me1/2, H3K56me2, and H3K79me1/2) and histone H4 residues (H4K20me1/2, H4R23me1, H4K31me2, H4R35me1/2, H4R36me1, H4R55me1 and H4K77me1) were increased in CS-exposed mouse lungs [32]